i still say there is probably enough diversity within the PHA gene which may impart positive characteristics. carriers may turn out to be the one's with the variation, other than the one that imparts the defect. not using carrier genetics may be an unfortunate constriction on diversity. hopefully it is clear i am not advocating perpetuating the defect itself, and the phenotype it may impart in a carrier status. this, unfortunately is one of those cases where enough sequencing of the gene from carriers, and how to use the data, with enoung individuals sequenced will never happen. sad. some people are fortunately still trying, and are testing individuals, and are marketing only clean animals with the phenotype they are looking for in a free package. it is moving in the direction it needs to, but it doesn't need to be completey elminated just yet, but only used for research purposes.